The following is a list of Lyme Disease research studies:
1. Peacock, B. N., Gherezghiher, T. B., Hilario, J. D., & Kellermann, G. H. (2015). New insights into Lyme disease. Redox biology, 5, 66-70. Abstract conclusion: Mitochondrial superoxide levels were significantly higher (p<0.0001) in Lyme borreliosis patients (n=32) as compared to healthy controls (n=30). Significantly low (p<0.0001) levels of cytosolic ionized calcium were also observed in Lyme borreliosis patients (n=11) when compared to healthy controls (n=11). These results indicate that there is an imbalance of reactive oxygen species and cytosolic calcium in Lyme borreliosis patients. The results further suggest that oxidative stress and interrupted intracellular communication may ultimately contribute to a condition of mitochondrial dysfunction in the immune cells of Lyme borreliosis patients.
2. Halperin, J. J., Little, B. W., Coyle, P. K., & Dattwyler, R. J. (1987). Lyme disease Cause of a treatable peripheral neuropathy. Neurology, 37(11), 1700-1700. Abstract conclusion: This neuropathy, which is quite different from the infrequent peripheral nerve syndromes previously described in this illness, is commonly present in late Lyme disease. This neuropathy presents with intermittent paresthesias without significant deficits on clinical examination and is reversible with appropriate antibiotic treatment. Neurophysiologic testing provides a useful diagnostic tool and an important measure of response to treatment.
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